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Fellow Julia Turner
Fellow

Dr Julia Turner

Official Fellow; Director of Studies; Official Fellow in Pathology ; Director of Studies in Preclinical Medicine ; Director of Studies in Pathology

Contact
Jmt18@cam.ac.uk⁠ 01223 762226⁠

Julia Turner is an alumna of New Hall (now Â鶹ƵµÀ), as an undergraduate in Natural Sciences and a PhD student in Molecular Oncology. She has worked as a postdoctoral scientist and principal investigator in academia at the University of California, San Francisco and Department of Pathology, University of Cambridge, and industry at Glaxo Wellcome UK Ltd. Julia was a fellow of New Hall from 1991-1996 and returned to the fellowship in 2019, taking up the post of Director of Studies in Preclinical Medicine, while supervising second and third year MVST and NST students in pathology

Research Interests 

My research career was as an immunologist, focusing on the control of T cell division during protective and pathogenic responses. 

Degrees 

  • PhD in Molecular Oncology, University of Cambridge
  • MA, BA in Natural Sciences, University of Cambridge

Authored work

  • Fehérvári, Z., Cooke, A., Brett, S. and Turner, J. (2002). Perturbation of naive TCR transgenic T cell functional responses and upstream activation events by anti-CD4 monoclonal antibodies. Eur. J. Immunol., 32: 333–340.

     

    Metcalfe S1, Alexander D, Turner J. (1994). FK506 and cyclosporin A each inhibit antigen-specific signalling in the T cell line 171 in the absence of a calcium signal. Cell Immunol. Oct 1;158(1):46-58. 

     

    Turner, JM (1993). IL-2-dependent induction of G1 cyclins in primary T cells is not blocked by rapamycin or cyclosporin A. International immunology, 5(10), 1199-1209. 

     

    Glaichenhaus, N., Shastri, N., Littman, D.R. & Turner, JM (1991) Requirement for association of p56lck with CD4 in antigen-specific signal transduction in T cells. Cell 64, 511-520. 

     

    Turner, JM Brodsky, M.H., Irving, B.A., Levin, S.D., Perlmutter, R.M. & Littman, D.R. (1990) Interaction of the unique amino-terminal region of the tyrosine kinase p56lck with the cytoplasmic domains of CD4 and CD8 is mediated by cysteine motifs. Cell 60, 755-765. 

Subject
Medicine